Power of Exile

The Power of Exile -
 Autism, A journey to recovery


Introduction: Sara’s Diet
and the IDEA


  1. Sara
  2. Sandra
  3. The Journey begins
  4. Sara joins our Family
  5. Journal Notes
  6. Impressions
  7. Influential People
  8. Center Stage
  9. I believe in Miracles
  10. Miracles in Abundance
  11. A Second Rainbow
  12. Widening Horizons
  1. World travel on a Wing and a Prayer
  2. Asperger Syndrome (Sam’s story)
  3. Autism: a Causal Theory and Treatment Option
  4. A Change in the Weather
  1. Second Timothy
  2. Turning Blue
  3. Food Intolerance in autism
  4. Sara’s Diet
    1. Introduction to the restricted diet
    2. Essential nutrients from foods
    3. Practical help with implementing a diet program
  5. What is Lutein?
  6. Autism, Pigments and the Immune System
  7. South Africa, World Community Autism Program
  8. Eating disorder in autism
  9. Autism, Origin – A Plausible Theory
  10. Autism, putting it all together

From: Autism, putting it all together
Female to male ratio re-evaluated
Since the majority of epidemiological research has been on the basis of the DSM-IV criteria, the current epidemiological information is inadequate to determine how many people have an autism spectrum disorder. What we do know is that the incidence of autism is reported at a much higher rate in males, and yet is often more severe in females, leading to a great deal of research into autism as a genetic condition. Using the definition of autism that we have given, specifically identifying the substance which is the target of the cytokine reaction to be lutein, we could foresee a rigorous attempt to compile information leading to the understanding that a female fetus with this condition is at greater risk of being naturally aborted. Information does exist which identifies female autists as having greater mortality risk factors. The in utero instance of the immune reaction that results in autism is likely to be equally distributed between male and female fetuses. Respiratory pigments released from the fetus would contribute to a higher incidence of toxic fetal environment, contributing to the possibility of spontaneous abortion. Spontaneous abortion has been identified as a potential outcome for a fetus with the CAG repeat. Also, the incidence of spontaneous abortion is identified as a greater potential risk factor for females generally.

The child’s relationship to parent/caregiver
Immune regulation and reduced enzyme production could contribute to abnormal levels of hallucinogenic by-products and naturally manufactured opioid by-products for the autist. Vitamin and mineral levels could impact the types of by-products produced. Direct sunlight exposure could also increase immune system responses in some autists. The resulting disturbance in the adrenocorticotropin–cortisol axis (ACTH/cortisol axis) - the immune system battling with the ‘molecules of emotion’ - leads to many of the challenging behaviours seen in autism.
  We know that extreme lack of nurturing can result in behaviourally challenging and learning-disabled children such as the Romanian orphans, similar in many ways to autists. We also know from experience that it is not likely that treating chemically dependant people will result in a positive outcome until the toxins (drugs, alcohol) are cleared from the body and we can therefore anticipate that it is not likely that we can expect positive outcome for autists until the chemical irregularities are understood and addressed.
  The child’s relationship to parent/caregiver is affected as the adult attempts to feed the infant/toddler foods which trigger adverse reactions and the toddler begins to recognize foods which make them feel good i.e. wheat and dairy, and avoids foods which make them feel bad i.e. coloured fruits and vegetables. The child craves nutrients lacking from the self-selective diet, and this can contribute to pica (ingesting sunscreens containing cocoa butter or aloe vera gel, mud, wall plaster, feces). The child does not develop a trust relationship with caregiver/parent. The young child is intoxicated from self-manufactured substances: opioids, gut fermentation products, bufotenin. The child is unable to proceed naturally through the developmental stages.
  Changes in eye colour have been reported and observed when a lutein-restricted diet was implemented: in the original study group of 800 individuals, this result occurred in about 47% of cases, but changes were not as easily recognized in the brown-eyed children. These changes included temporary ‘blue sclera’ responses.

Qualitative impairment in social interaction
As we look at the autist through ‘new eyes’, the characteristic presentation of qualitative impairment in social interaction would be an expected outcome for infants who experience environmental stimuli through the primal immune response of survival while simultaneously experiencing the Major Histamine Class (MHC) delayed hypersensitivity type IV reactions or Type V cytokine initiated cytokine mediated responses.

Other factors then contribute significantly to the arrested developmental process for the autist:
  1. Immune system manufacture of ‘natural pain killers’ resulting in infants who are under the influence of self-manufactured opioids, gut fermentation products, hallucinogens from abnormal tryptophan and cytochrome metabolism;
  2. ‘Fight or flight’ response to internal and environmental stimuli;
  3. Medical interventions i.e. vaccines and antibiotics resulting in a variety of outcomes for individuals including vaccine reactions and gut dysbiosis;
  4. Inability to develop emotional maturity skills as a result of sensory difficulties, lack of trust, reduced capacity for success when attempting simple tasks;
  5. Diet, providing the immune trigger, exacerbating the immune response and which may result in unrecognised pain stimuli in the autist from the breakdown of the food pathogen and decreased potential for producing prostaglandin type pain regulators.
The individual with autism may undergo additional insults:
  1. Toxins build up in a metabolism with reduced capacity for xenobiotic and hepatic metabolite removal and/or increase in hepatic metabolite manufacture;
  2. Self-selective diets result in nutrient excesses and deficiencies and opioid addiction. These factors contribute to acetylcholine depletion (affecting speech, behaviour, motor control, abstract thinking, memory and speech) and arachidonic acid and cytochrome P-450 responses resulting in symptoms some of which are perceived as behaviours in this population.
  3. Conditions, which co-occur with autism, are not detected, tested for or treated.
  4. Hyperactivity as toxins reach the brain. (The brain healing process, or ‘re-growth of mossy fibers’, results in hyperactivity.)
  5. Increased presentation of skin pigmentation abnormalities and immune epithelial output resulting in itching, scratching and contributing to self-injurious behaviours. This can be reduced or eliminated when the immune trigger(s) are identified and removed from the diet;
  6. Altered response to ultraviolet light exposure further affecting hormone balance and sometimes contributing to behavioural-type responses. This response can be reduced with protective eye-wear and protective clothing;
  7. Self-injurious behaviour, which may contribute to the production of natural pain killers (opioid type) and could therefore be understood as an attempt to self medicate rather than to self injure;
  8. Gut disturbance and continence difficulties potentially initiating oppositional-defiant type behaviours surrounding feeding and toilet training;
  9. Food intolerances and abnormal serotonin metabolism contribute to the development of eating disorders ranging from anorexia to compulsive eating (serotonin contributes to regulating feelings of satiation);
  10. Visual and auditory processing differences 119 seen as ‘tuning out’ behavior and reflected in the resulting stereotypical behaviors;
  11. Mitochondrial abnormalities 57  resulting in decreased cellular energy for some, appearing as laziness.